Efecto de la adición de 18:0 a una dieta rica en aceite de pescado sobre el rendimiento productivo y el perfil de ácidos grasos de la leche de ovejas lecheras

• P.G. Toral ^[1] ; G. Hervás ^[1] ; D. Carreño ^[1] ; J.S. Gonzalez ^[1] ; J. Amor ^[1] ; P. Frutos ^[1]
  1. [1] CSIC-ULE
• Localización: XVI Jornadas sobre Producción Animal: 19 y 20 de mayo de 2015, Zaragoza / Javier Álvarez Rodríguez (aut.), Begoña Panea Doblado (aut.), Jorge Hugo Calvo Lacosta (aut.), Mireia Blanco Alibés (aut.), José Alfonso Abecia Martínez (aut.), Daniel Villalba Mata (aut.), María Ángeles Latorre Górriz (aut.), Vol. 1, 2015, ISBN 978-84-606-7969-1, págs. 161-163
• Idioma: español
• Títulos paralelos:
  • Effect of the addition of 18:0 to a diet rich in fish oil on animal performance and milk fatty acid profile in dairy ewes
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  • Texto Completo Libro
• Resumen

  • Diet supplementation with fish oil (FO) modulates the fatty acid (FA) profile of sheep milk but induces milk fat depression (MFD). This response has been associated with a shortage of 18:0 for mammary cis-9 18:1 synthesis (through ǻ-desaturation) and its potential impact on the maintenance of milk fat fluidity. This study was, therefore, conducted to test the hypothesis that supplemental 18:0 would alleviate FO-induced MFD in ewes. The experiment followed a 3×3 Latin square design (4 animals/group) with 3 periods of 4 weeks each and 3 experimental diets: non-supplemented, supplemented with 2% FO or 2% FO plus 2% 18:0. Afterwards, the digestibility of additional 18:0 was measured using 6 lactating sheep. Compared with the control, both supplemented diets reduced milk fat content in a similar proportion (20%), suggesting that the inclusion of 18:0 does not alleviate FO-induced MFD. This result cannot be fully explained by the relatively low digestibility coefficient of additional 18:0, although this supplement partially compensated for the decrease in milk cis-9 18:1 concentration associated with FO intake. Changes in the proportion of odd- and branchedchain and biohydrogenation-derived FA (which suggest, for example, a shift from trans-11 to trans-10 18:1) might contribute to explain the unsuccessful response to the addition of 18:0.